B.S. University of Science & Technology of China
Ph.D. University of Illinois at Urbana-Champaign
Postdoc. The Rockefeller University
Molecular mechanism of mammalian autophagy and its relevance to human diseases; neurodegeneration and aging; proteomics.
Autophagy is a lysosomal degradation pathway that plays important roles in a variety of essential cellular processes and many devastating human diseases including cancer, infection, liver disease, myopathy and neurodegeneration. The Wang lab takes an integrated approach that combines mouse genetics, proteomics, biochemistry and cell biology to study (i) all key steps of the mammalian autophagy pathway including signaling and regulation, (ii) roles of autophagy in the healthy brain and that undergoes neurodegeneration, (iii) roles of autophagy in aging; and (iv) roles of autophagy in other human diseases. The Wang lab is open to the IBS student rotation and thesis work.
NIH Center of Biomedical Research Excellence (COBRE) in the Molecular Basis of Human Disease, 9/2009-6/2014
Ellison Medical Foundation New Scholar in Aging, 8/2010-7/2014 (see an interview of Dr. Wang by UKNow http://uknow.uky.edu/content/qingjun-wang-receives-new-scholar-award)
University Research Postdoctoral Fellowship, 1/2011-12/2012 (Awardee: Deanna H. Morris)